時(shí)　間: 下午13:30—15:00

日　期:  2018年9月4日（周二） 

地　點(diǎn): 心理所南樓六層會(huì )議室 

摘 要： 

　　Catecholamine (CA) neurotransmitters (e.g., dopamine) have long been implicated playing a critical role in executive control and decision making. Recently, evidence from neurodegenerative patients and healthy population suggested that CA also modulates language processing. However, the question of what kind of influence the CA might exert on language is still open. My recent study investigated this question by examining the effects of methylphenidate (MPH), which is a CA agonist, on EEG response related to language processing. The results demonstrated a clear neuropharmacological effect that CA neurotransmitters provide a relevance signal that enhances semantic integration operations, regardless of whether language processing is goal-relevant or not. The Basal Ganglia projections to Frontal Cortex might mediate the language processing effects of these neuropharmacological agents. These results contradicted the general hypothesis that CA promotes goal-relevant processes, while inhibiting goal-irrelevant information, suggesting that our capacity for language is a core system rooted in our biological system. 

報告者簡(jiǎn)介： 

　　譚瀅瀅博士2008年本科畢業(yè)于北京大學(xué)心理學(xué)系，2015年獲得美國Rice University心理學(xué)系認知神經(jīng)科學(xué)博士學(xué)位，現在荷蘭馬克思普朗克研究所心理語(yǔ)言學(xué)中心從事博士后研究。主要研究方向是人類(lèi)語(yǔ)言加工的認知神經(jīng)基礎，比如語(yǔ)言加工與工作記憶和認知控制功能的交互影響，以及特定神經(jīng)藥物干預對語(yǔ)言加工的影響。主要研究方法包括結合EEG技術(shù)和使用心理藥理學(xué)手段，測量個(gè)體認知行為能力差異，考察健康被試和腦損傷病人（失語(yǔ)癥患者）的語(yǔ)義語(yǔ)法系統加工，與認知障礙的認知基礎。